Tricuspid valve dysplasia(TVD) in an American cocker spaniel dog

American cocker spaniel dog에서 발생한 삼첨판 이형성 증례

  • Park, Chul (Department of Veterinary Internal Medicine, College of Veterinary Medicine, Konkuk University) ;
  • Choi, Chi-Bong (Department of Veterinary Internal Medicine, College of Veterinary Medicine, Konkuk University) ;
  • Sur, Jung-Hyang (Department of Veterinary Pathology, College of Veterinary Medicine, Konkuk University) ;
  • Chung, Byung-Hyun (Department of Veterinary Internal Medicine, College of Veterinary Medicine, Konkuk University) ;
  • Park, Hee-Myung (Department of Veterinary Internal Medicine, College of Veterinary Medicine, Konkuk University)
  • 박철 (건국대학교 수의과대학 내과학 교실) ;
  • 최치봉 (건국대학교 수의과대학 내과학 교실) ;
  • 서정향 (건국대학교 수의과대학 병리학 교실) ;
  • 정병현 (건국대학교 수의과대학 내과학 교실) ;
  • 박희명 (건국대학교 수의과대학 내과학 교실)
  • Accepted : 2004.02.24
  • Published : 2004.03.31

Abstract

A 2-year-old, female, American cocker spaniel dog presented for a 1-year history of severe ascites, exercise intolerance, tachypnea. At that time, she was in an emergency state. First, the dog was stabilized with oxygen therapy. A diagnosis of cardiac problem was made from history, auscultation, radiograph, ECG, and echocardiography. Jugular pulsation was palpated and a harsh, systolic murmur of tricuspid regurgitation was prominent at the right cardiac apex. Tricuspid valve dysplasia (TVD) was confirmed with echocardiography, accompanying enormous myocardial hypertrophy. The clinical signs had been improved for 8 months with careful therapy and periodic abdominocentesis, and ascites was well controlled. The situation, however, became worse quickly in a week because the client did not follow our management schedule. Finally, she died due to dyspnea and shock. After the spontaneous death, necropsy and histopathological examination were performed and when we opened the thorax, a significantly large heart was observed. On histopathological findings, grossly myocardium appeared pale initially, then progressed to yellow and white. Microscopically, there was an extensive hemorrhage along with loss of myocardial striations. Interstitial fibrosis and various degenerative alterations in myocytes were also present.

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