• Title/Summary/Keyword: Evoked potentials%2C somatosensory

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Do N37 and P37 Potentials Have Different Generators in Somatosensory Evoked Potential? - Analysis Using Gating Mechanism - (체성감각 유발전위에서 N37과 P37은 다른 발생기를 가지고 있는가? - gating 현상을 이용한 분석 -)

  • Park, Young Seok;Cha, Jae Kwan;Kim, Sang Ho;Kim, Jae Woo
    • Annals of Clinical Neurophysiology
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    • v.1 no.2
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    • pp.106-111
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    • 1999
  • Backgroud : The generators of N37 and P37 of posterior tibial nerve somatosensory evoked potential(PTSEP) have not been exactly known. Recently, some reports suggested that P37 and N37 might have different generator. We conducted a study to know the generators of P37 and N37 of PTSEP using gating mechanism. Methods : We evaluated subcortical and cortical somatosensoy evoked potentials(SEPs) in response to posterior tibial nerve stimulation in 3 experimental conditions of foot movement and compared them with PTSEPs in full relaxation of foot. The experimental conditions were: (a) active flexion-extention of stimulated foot, (b) isometric contraction of the stimulated foot, (c) passive flexion-extention of the stimulate foot. We analyzed the latencies and amplitudes of following potentials; P30, N37, P37, and N50. Results : The amplitude of P30 potential did not change during at any paradigms. The amplitudes of P37 and N50 were significantly attenuated in all condition. However, the amplitude of N37 showed no significant change during at any paradigms. Conclusions : These results suggest that the generators of P37 and N37 of PTSEP be different in cortex.

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Changes in Dermatomal Somatosensory Evoked Potentials according to Stimulation Intensity and Severity of Carpal Tunnel Syndrome

  • Sohn, Soo-Youn;Seo, Jeong-Hwan;Min, Yong;Seo, Min-Ho;Eun, Jong-Pil;Song, Kyung-Jin
    • Journal of Korean Neurosurgical Society
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    • v.51 no.5
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    • pp.286-291
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    • 2012
  • Objective : To investigate the change of latency of cervical dermatomal somatosensory evoked potential (DSEP) according to stimulation intensity (SI) and severity of carpal tunnel syndrome (CTS). Methods : Stimulation sites were the C6, C7, and C8 dermatomal areas. Two stimulation intensities $1.5{\times}$sensory threshold (ST) and $2.5{\times}ST$ were used on both normal and CTS patients. Results : In moderate CTS, the latencies of C6 and C7 DSEP during $1.5{\times}ST$ SI and those of C7 DSEP during $2.5{\times}ST$ SI were significantly delayed compared with the values of normal subjects. Significant correlation between the latency of C7 DSEP of $2.5{\times}ST$ stimulation and the median sensory nerve conduction velocity was observed. Conclusion : We suggest that these data can aid in the diagnosis of cervical sensory radiculopathy using low stimulation intensity and of those who have cervical sensory radiculopathy combined with CTS patients.

The quantitative sensory testing is an efficient objective method for assessment of nerve injury

  • Kim, Young-Kyun;Yun, Pil-Young;Kim, Jong-Hwa;Lee, Ji-Young;Lee, Won
    • Maxillofacial Plastic and Reconstructive Surgery
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    • v.37
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    • pp.13.1-13.7
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    • 2015
  • Background: This study evaluated Somatosensory evoked potentials (SEP), Quantitative sensory testing (QST), and thermography as diagnostic methods for nerve injury. Methods: From 2006 through 2011, 17 patients (mean age: 50.1 years) from ${\bigcirc}{\bigcirc}{\bigcirc}{\bigcirc}$ Hospital who sought care for altered sensation after dental implant treatment were identified. The mean time of objective assessment was 15.2 months after onset. Results: SEP of Inferior alveolar nerve(IAN) was $15.87{\pm}0.87ms$ on the normal side and $16.18{\pm}0.73ms$ on the abnormal side. There was delayed N20 latency on the abnormal side, but the difference was not statistically significant. In QST, the abnormal side showed significantly higher scores of the current perception threshold at 2 KHz, 250 Hz, and 5 Hz. The absolute temperature difference was $0.55^{\circ}C$ without statistically significance. Conclusion: These results indicate that QST is valuable as an objective method for assessment of nerve injury.