• Title/Summary/Keyword: average cell delays

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Performance Analysis of Output Queued Batcher-Banyan Switch for ATM Network (ATM 망에 적용 가능한 출력단 버퍼형 Batcher-Banyan 스위치의 성능분석)

  • Keol-Woo Yu;Kyou Ho Lee
    • Journal of the Korea Society for Simulation
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    • v.8 no.4
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    • pp.1-8
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    • 1999
  • This paper proposes an ATM switch architecture called Output Queued Batcher-Banyan switch (OQBBS). It consists of a Sorting Module, Expanding Module, and Output Queueing Modules. The principles of channel grouping and output queueing are used to increase the maximum throughput of an ATM switch. One distinctive feature of the OQBBS is that multiple cells can be simultaneously delivered to their desired output. The switch architecture is shown to be modular and easily expandable. The performance of the OQBBS in terms of throughput, cell delays, and cell loss rate under uniform random traffic condition is evaluated by computer simulation. The throughput and the average cell delay are close to the ideal performance behavior of a fully connected output queued crossbar switch. It is also shown that the OQBBS meets the cell loss probability requirement of $10^{-6}$.

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A CMOS Cell Driver Model to Capture the Effects of Coupling Capacitances (결합 커패시턴스의 영향을 고려한 CMOS 셀 구동 모델)

  • Cho, Kyeong-Soon
    • Journal of the Institute of Electronics Engineers of Korea SD
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    • v.42 no.11
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    • pp.41-48
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    • 2005
  • The crosstalk effects that can be observed in the very dee submicron semiconductor chips are due to the coupling capacitances between interconnect lines. The accuracy of the full-chip timing analysis is determined by the accuracy of the estimated propagation delays of cells and interconnects within the chip. This paper presents a CMOS cell driver model and delay calculation algerian capturing the crosstalk effects due to the coupling capacitances. The proposed model and algorithm were implemented in a delay calculation program and used to estimate the propagation delays of the benchmark circuits extracted from a chip layout. We observed that the average discrepancy from HSPICE simulation results is within $1\%$ for the circuits with a victim affected by $0\~10$ aggressors.

The Macroscopic Model for Signalized Intersections to Consider Progression in relation to Delay (지체시간과 연동성을 동시에 고려하는 신호교차로 시뮬레이션 모형의 개발)

  • Han, Yohee;Kim, Youngchan
    • The Journal of The Korea Institute of Intelligent Transport Systems
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    • v.11 no.6
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    • pp.15-22
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    • 2012
  • A performance index of singalized intersections is a standard to optimize signal control variables and to manage traffic flow. Traffic delays is generally used to minimize the average delay time on intersections or networks, progression efficiency is used to improve travel speed of main cooridors or to provide transit signal priority. We manage traffic flows with only selecting one index between delays and progression according to the objective of traffic management and field characteristics. In real field, the driver's satisfaction is high in any performance criteria when the waiting time is shorter and the unnecessary stop in front of traffic is smaller. This paper aims to develop simulation model to represent real progression with concurrently considering delays and progression. In order to reflect an effect of level of traffic volumes and residual queues which don't be considered in prior progression model, we apply shockwave model with flow-density diagram. We derive Cell Transmission Model of Daganzo in order to develop the delay index and the progression index for the macroscopic simulation model. In order to validate the effect, we analysis traffic delays and progression efficiency with comparing this model to Transyt-7F and PASSER V.

Korean Red Ginseng Significantly Slows CD4 T Cell Depletion over 10 Years in HIV-1 Infected Patients: Association with HLA

  • Cho, Young-Keol;Sung, Heungsup;Kim, Tai Kyu;Lim, Ji Youn;Jung, You Sun;Kang, Sang-Moo
    • Journal of Ginseng Research
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    • v.28 no.4
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    • pp.173-182
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    • 2004
  • We have shown that long-term intake of Korean red ginseng (KRG) delays disease progression in HIV-I infected patients. In the present study to investigate whether this slow progression was associated with protective human leukocyte antigen (HLA) alleles as well as with KRG-intake, we have performed clinical analysis of 31 HIV-1 infected patients who have been living for more than 10 years without any antiretroviral therapy. Average amount of KRG-intake over $130\;{\pm}16$ months was $4,797\;{\pm}4,921\;g$ and the annual decrease in CD4 T cell (AD) was $30\;{\pm}29{\mu}L$. We observed significant correlations among amount of KRG-intake, AD(r=-0.53, P < 0.01), and plasma HIV-1 RNA copy (r=-0.35, P < 0.05), along with a significant correlation between KRG-intake and HLA score AD(r=-0.49, P < 0.01), whereas there was no significant correlation between HLA score and AD or viral load. When the 31 patients were divided into 2 groups based on the amount of KRG-intake, the $AD(14/{\mu}L)$ in the 16 patients who had taken higher amounts of KRG was significantly less than that $(49/{\mu}L)$ in the 15 patients with a little or no KRG-intake (P < 0.01). These data indicate that KRG-intake sig­nificantly slows CD4 T cell depletion in HIV-1 infected patients.

Prophylactic cranial irradiation in limited small-cell lung cancer : incidence of brain metastasis and survival and clinical aspects (예방적 두강내 방사선 조사후 소세포 폐암 환자의 뇌전이 빈도와 생존율에 대한 연구)

  • Suh, Jae-Chul;Kim, Myung-Hoon;Park, Hee-Sun;Kang, Dong-Won;Lee, Kyu-Seung;Ko, Dong-Seok;Kim, Geun-Hwa;Jeong, Seong-Su;Cho, Moon-June;Kim, Ju-Ock;Kim, Sun-Young
    • Tuberculosis and Respiratory Diseases
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    • v.49 no.3
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    • pp.323-331
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    • 2000
  • Purpose: Brain metastases are present in approximately 10-16% of small cell lung cancer patients at diagnosis. Brain metastasis is an important clinical problem associated with increasing the survival rate, with a cumulative incidence of up to 80% in patients surviving 2 years. Prophylactic cranial irradiation(PCI) reduces the incidence of brain matastasis and may prolong survival in patients with limited small-cell lung cancer who achieved complete remission. This study was performed to analyze the incidence of brain metastasis, survival and clinical aspects after PCI in patients with limited small-cell lung cancer who achieved complete remission. Methods : Between 1989 and 1999, forty-two patients with limited small-cell lung cancer who achived achieved complete remission after therapy were enrolled into this study retrospectively. All patients received etoposide and cisplatin(VPP) alternating with cytoxan, adriamycin, and vincristine(CAV) every 3 weeks for at least 6 cycles initially. All patients received thoracic radiotherapy: concurrent(38.1%) and sequential(61.9%). All patients received late PCI. Results : Most patients(88.1%) were men, and the median age was 58 years. The median follow-up duration was 18.1 months. During the follow-up period, 57.1% of the patients developed relapse. The most frequent site of relapse was chest(35.7%), followed by brain(14.3%), liver(11.9%), adrenal gland(44%), and bone(2.2%). With the Kaplan-Meier method, the average disease-free interval was 1,090 days(median 305 days). The average time to development of brain relapse after PCI and other sites relapse(except brain) were 2,548 days and 1,395 days(median 460 days), respectively. The average overall survival was 1,233 days(median 634 days, 21.1 months), and 2-year survival rates was 41.7%. The average overall survival in the relapse group was 642 days(median 489 days) and in the no relapse group was 2,622 days(p<0.001). The average overall survival in the brain relapse group was 928 days(median 822 days) and in the no brain relapse group was 1,308 days(median 634 days)(p=0.772). In most patients(85.7%), relapse(except brain) or systemic disease was the usual cause of death. Brain matastasis was the cause of death in 14.3% of the cases. Conclusions : We may conclude that PCI reduces and delays brain metastasis in patients with limited small cell lung cancer who achieved complete remission. We found decreased survival in relapse group but, no significant survival difference was noted according to brain matastasis. And relapse(except brain) or systemic disease was the usual cause of death. In order to increase survival, new treatment strategies for control methods for relapse and systemic disease are required.

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Unfairness of Congestion Control for Point-to-Multipoint Connections on ATM (ATM 상의 다중점 연결을 위한 폭주 제어 기법의 불공정성)

  • Choi, Won-Jeong;Lee, Mee-Jeong
    • The Transactions of the Korea Information Processing Society
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    • v.5 no.5
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    • pp.1311-1319
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    • 1998
  • The methods of providing available bandwidth adaptively using feedback to maximize the utilization of network as well as the quality of service have been the focus of recent research activities for ATM(Asynchronous Transfer Mode). This study has been extended from the point-to-point connection to a point-to-multipoint environment as the number of applications requiring multicast service increases. It is known that the effectiveness of feedback based congestion control scheme diminishes as propagation delay increases. Especially for a multicast connection consisting of various paths and destinations with different performance and congestion status, the problem of unfairness due to different propagation delays may occur. The degree of such unfairness may change depending on various aspects of congestion control schemes. These has been, however, relatively little study on these problems. In this paper, we present how various aspects of control schemes-length of the interval between feedback generations, point of time to coalesce feedback cells from child paths, decreasing factor of source rate in case of congestion-affect the degree of unfairness. Simulation results show that degree of unfairness changes according to when the feedback coalescing happens. Expecially it is shown that the effect of feedback coalescing time to the degree of unfairness is more significant for the smaller feedback interval. It is also found that as the source rate decreasing factor becomes larger the average ACR(Allowed Cell Rate) at the source gets lower and the degree of unfairness grow larger.

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